Abstract | BACKGROUND: METHODS: RESULTS: CONCLUSIONS: These results indicate that ATX treatment is neuroprotective against SAH, possibly through suppression of cerebral inflammation.
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Authors | Xiang-Sheng Zhang, Xin Zhang, Qi Wu, Wei Li, Chun-Xi Wang, Guang-Bin Xie, Xiao-Ming Zhou, Ji-Xin Shi, Meng-Liang Zhou |
Journal | The Journal of surgical research
(J Surg Res)
Vol. 192
Issue 1
Pg. 206-13
(Nov 2014)
ISSN: 1095-8673 [Electronic] United States |
PMID | 24948541
(Publication Type: Journal Article, Research Support, Non-U.S. Gov't)
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Copyright | Copyright © 2014 Elsevier Inc. All rights reserved. |
Chemical References |
- Interleukin-1beta
- NF-kappa B
- Neuroprotective Agents
- Tumor Necrosis Factor-alpha
- Xanthophylls
- astaxanthine
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Topics |
- Animals
- Blood-Brain Barrier
(drug effects)
- Brain Edema
(drug therapy, immunology, metabolism)
- Cell Death
(drug effects)
- Disease Models, Animal
- Interleukin-1beta
(metabolism)
- Male
- NF-kappa B
(metabolism)
- Neuritis
(drug therapy, immunology, metabolism)
- Neuroprotective Agents
(pharmacology)
- Optic Chiasm
(drug effects, immunology, metabolism)
- Rats, Sprague-Dawley
- Subarachnoid Hemorrhage
(drug therapy, immunology, metabolism)
- Tumor Necrosis Factor-alpha
(metabolism)
- Xanthophylls
(pharmacology)
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