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N- (5- (2- chlorobenzyl)thiazole- 2- yl)benzofuran- 2- carboxamide

structure in first sopurce

Also Known As:

HS-111

Networked: 3 relevant articles (0 outcomes, 0 trials/studies)

Bio-Agent Context: Research Results

Heterocyclic Compounds: 198
- 1-Ring Heterocyclic Compounds
  - Azoles: 2138
    - Thiazoles: 392
      - N-(5-(2-chlorobenzyl)thiazole-2-yl)benzofuran-2-carboxamide: 3
- Fused-Ring Heterocyclic Compounds
  - 2-Ring Heterocyclic Compounds
    - Benzofurans: 26
      - N-(5-(2-chlorobenzyl)thiazole-2-yl)benzofuran-2-carboxamide: 3
Organic Chemicals: 133
- Sulfur Compounds: 278
  - Thiazoles: 392
    - N-(5-(2-chlorobenzyl)thiazole-2-yl)benzofuran-2-carboxamide: 3

Experts

1.	Choi, Myung-Joo: 1 article (03/2012)
2.	Hong, Soon-Sun: 1 article (03/2012)
3.	Hong, Sungwoo: 1 article (03/2012)
4.	Jung, Kyung Hee: 1 article (03/2012)
5.	Kim, Donghee: 1 article (03/2012)
6.	Lee, Hyunseung: 1 article (03/2012)
7.	Lee, Ju-Hee: 1 article (03/2012)
8.	Akbari, Mohammad T: 1 article (01/2009)
9.	Arab, Aida: 1 article (01/2009)
10.	Hamid, Mohammad: 1 article (01/2009)

Related Diseases

1.	beta-Thalassemia (Cooley's Anemia) 11/01/2007 - "The HS-111 (-21 G>A) and 3'HS1 (+179 C>T) transitions form CH haplotypes, which occur at different frequencies in beta-thalassemia intermedia and major patients and normal (nonthalassemic) individuals. " 01/01/2009 - "Molecular analysis of gamma-globin promoters, HS-111 and 3'HS1, in beta-thalassemia intermedia patients associated with high levels of Hb F." 01/01/2009 - "The nucleotide (nt) variations in the promoter region of the gamma-globin genes, HS-111 and 3'HS1 regions, were studied in Iranian patients with beta-thalassemia intermedia (beta-TI), beta-thalassemia major (beta-TM) and healthy individuals. "
2.	Hepatocellular Carcinoma (Hepatoma) 03/01/2012 - "The effect of HS-111, a novel thiazolamine derivative, on apoptosis and angiogenesis of hepatocellular carcinoma cells."
3.	Hypoxia (Hypoxemia) 03/01/2012 - "In addition, HS-111 decreased protein expression of hypoxia-inducible factor (HIF-1α) and secretion of vascular endothelial growth factor (VEGF), and inhibited tube formation and the migration of human umbilical vein endothelial cells (HUVECs). "

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