Abstract | INTRODUCTION AND HYPOTHESIS: METHOD: We performed a register-based national cohort study of primipara who delivered between 1985 and 1988 and had no further deliveries. Medical Birth Register data were linked to data from postal questionnaires distributed 20 years after the birth (response rate 65.2 %, n = 5 236). Main outcome measures were prevalence and risk factors for pelvic floor disorders (PFDs) and OASI and their impact after VE compared with SVD and ACS. Multivariate logistic regression models were used. RESULTS: The late prevalence of UI, sPOP, and FI was almost identical between VE and SVD. VE almost tripled the rate of OASI compared with SVD (6.3 vs. 2.4 %, p < 0.001). FI rate after an OASI was similar for both VE and SVD [30.2 vs. 27.8 %, adjusted odds ratio (aOR) 1.12; 95 % confidence interval (CI) 0.49-2.56]. Comparing VE without laceration with VE complicated by OASI increased the rate of FI (from 15.4 to 30.2 %, aOR 2.55; 95 % CI 1.26-5.15) and UI (from 39.0 to 61.4 %, aOR 2.28; 95 % CI 1.19-4.34), but the rate of sPOP was almost unaltered (from 15.0 to 15.9 %). CONCLUSIONS: VE did not result in additional long-term PFDs provided the rate of OASI was similar to that after SVD. OASI after VE substantially increased the prevalence of FI and UI but did not alter the rate of sPOP.
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Authors | Ida Nilsson, Sigvard Åkervall, Ian Milsom, Maria Gyhagen |
Journal | International urogynecology journal
(Int Urogynecol J)
Vol. 27
Issue 7
Pg. 1051-6
(Jul 2016)
ISSN: 1433-3023 [Electronic] England |
PMID | 26714984
(Publication Type: Journal Article)
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Topics |
- Adult
- Anal Canal
(injuries)
- Cohort Studies
- Fecal Incontinence
(enzymology, etiology)
- Female
- Humans
- Parity
- Pelvic Floor Disorders
(epidemiology, etiology)
- Pelvic Organ Prolapse
(epidemiology, etiology)
- Postoperative Complications
(epidemiology, etiology)
- Pregnancy
- Prevalence
- Sweden
(epidemiology)
- Urinary Incontinence
(epidemiology, etiology)
- Vacuum Extraction, Obstetrical
(adverse effects)
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