Abstract | BACKGROUND: METHODS: RESULTS: CONCLUSIONS: Our findings suggest that hyperuricemia contributes to an increase in serum concentration of allantoin, though they do not indicate that hyperuricemia is a major factor for controlling oxidative stress in vivo.
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Authors | Masafumi Kurajoh, Hidenori Koyama, Takuhito Shoji, Chihiro Sumida, Asako Yamamoto, Zenta Tsutsumi, Yuji Moriwaki, Tetsuya Yamamoto, Masafumi Koga |
Journal | International journal of clinical pharmacology and therapeutics
(Int J Clin Pharmacol Ther)
Vol. 50
Issue 4
Pg. 265-71
(Apr 2012)
ISSN: 0946-1965 [Print] Germany |
PMID | 22456297
(Publication Type: Journal Article, Research Support, Non-U.S. Gov't)
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Chemical References |
- Biomarkers
- Uricosuric Agents
- Uric Acid
- 8-epi-prostaglandin F2alpha
- Allantoin
- Benzbromarone
- 8-Hydroxy-2'-Deoxyguanosine
- Creatinine
- Dinoprost
- Deoxyguanosine
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Topics |
- 8-Hydroxy-2'-Deoxyguanosine
- Adult
- Allantoin
(blood)
- Benzbromarone
(therapeutic use)
- Biomarkers
(blood, urine)
- Case-Control Studies
- Creatinine
(urine)
- Deoxyguanosine
(analogs & derivatives, urine)
- Dinoprost
(analogs & derivatives, urine)
- Gout
(blood, drug therapy, urine)
- Humans
- Hyperuricemia
(blood, drug therapy, urine)
- Japan
- Male
- Middle Aged
- Oxidative Stress
(drug effects)
- Time Factors
- Treatment Outcome
- Uric Acid
(blood)
- Uricosuric Agents
(therapeutic use)
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