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BJ-2266
downregulates the JAK/STAT signaling pathway; structure in first source
Also Known As:
N-((5-hydroxy-3,4,6-trimethylpyridin-2-yl)carbamothioyl)benzamide
Networked:
1
relevant articles (
1
outcomes,
0
trials/studies)
Relationship Network
Bio-Agent Context: Research Results
Organic Chemicals: 133
Amines: 4871
Aminopyridines: 77
BJ-2266: 1
Amides: 2428
Urea: 15015
Thiourea: 362
BJ-2266: 1
Sulfur Compounds: 278
Thiourea: 362
BJ-2266: 1
Heterocyclic Compounds: 198
1-Ring Heterocyclic Compounds
Pyridines: 88
Aminopyridines: 77
BJ-2266: 1
Experts
1.
Chang, Jae-Hoon
: 1 article (01/2017)
2.
Jeong, Byeong-Seon
: 1 article (01/2017)
3.
Timilshina, Maheshwor
: 1 article (01/2017)
4.
You, Zhiwei
: 1 article (01/2017)
Related Diseases
1.
Experimental Autoimmune Encephalomyelitis (Encephalomyelitis, Autoimmune Experimental)
01/01/2017 - "
BJ-2266 ameliorates experimental autoimmune encephalomyelitis through down-regulation of the JAK/STAT signaling pathway.
"
01/01/2017 - "
We assessed the inhibitory effect of BJ-2266 in experimental autoimmune encephalomyelitis (EAE).
"
2.
Disease Progression
01/01/2017 - "
Our results suggest that BJ-2266 treatment significantly suppresses EAE disease progression with reduced generation of Th1 and Th17 cells.
"