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gastric inhibitory polypeptide (3-30)-amide

a GIP receptor antagonist

Also Known As:

GIP (3-30)NH2; GIP(3-30)NH2; glucose insulinotropic polypeptide(3-30)amide

Networked: 3 relevant articles (0 outcomes, 0 trials/studies)

Bio-Agent Context: Research Results

Experts

1.	Hartmann, Bolette: 3 articles (05/2021 - 01/2019)
2.	Christensen, Mikkel B: 2 articles (05/2021 - 01/2020)
3.	Gasbjerg, Lærke S: 2 articles (05/2021 - 01/2020)
4.	Holst, Jens J: 2 articles (05/2021 - 01/2020)
5.	Knop, Filip K: 2 articles (05/2021 - 01/2020)
6.	Rosenkilde, Mette M: 2 articles (05/2021 - 01/2020)
7.	Vilsbøll, Tina: 2 articles (05/2021 - 01/2020)
8.	Dela, Flemming: 1 article (05/2021)
9.	Jensen, Mette H: 1 article (05/2021)
10.	Krogh, Liva L: 1 article (05/2021)

Related Diseases

1.	Bone Resorption 01/01/2020 - "In conclusion, blockade of the GIPR with GIP(3-30)NH2 diminished GIP-induced CTX and P1NP responses, showing that these effects are GIPR-mediated and that GIPR antagonism might interfere with bone resorption." 05/21/2021 - "Suppression of bone resorption (assessed by circulating carboxy-terminal collagen crosslinks (CTX)) was impaired during GIP(3-30)NH2 infusion compared with placebo (ΔbsAUCCTX; ±s.e.m.; -4.9 ± 2 ng/mL × min, P = 0.005) corresponding to a ~50% reduction. " 01/01/2019 - "In rats, subchronic treatment with GIPR antagonist, rat GIP (3-30)NH2, did not modify food intake and bone resorption, but significantly increased body weight, body fat mass, triglycerides, LPL, and leptin levels compared with vehicle treated rats. "
2.	Body Weight (Weight, Body) 01/01/2019 - "In rats, subchronic treatment with GIPR antagonist, rat GIP (3-30)NH2, did not modify food intake and bone resorption, but significantly increased body weight, body fat mass, triglycerides, LPL, and leptin levels compared with vehicle treated rats. "

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