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Brain glucose metabolism in thalamic syndrome.

Abstract
Regional brain glucose metabolism was studied in a case of postischaemic thalamic syndrome. Despite a normal density of the thalamus on MRI and CT images, a 17% relative hypometabolism was found in the posterior thalamus on the affected side. This observation of functional anomalies in the posterior thalamic complex in case of thalamic syndrome is compatible with a deregulated processing of pain-related information at this level.
AuthorsE C Laterre, A G De Volder, A M Goffinet
JournalJournal of neurology, neurosurgery, and psychiatry (J Neurol Neurosurg Psychiatry) Vol. 51 Issue 3 Pg. 427-8 (Mar 1988) ISSN: 0022-3050 [Print] England
PMID3258903 (Publication Type: Case Reports, Journal Article)
Chemical References
  • Blood Glucose
  • Fluorodeoxyglucose F18
  • Deoxyglucose
Topics
  • Blood Glucose (metabolism)
  • Brain (pathology)
  • Cerebral Infarction (pathology)
  • Deoxyglucose (analogs & derivatives)
  • Female
  • Fluorodeoxyglucose F18
  • Humans
  • Magnetic Resonance Imaging
  • Middle Aged
  • Putamen (pathology)
  • Syndrome
  • Thalamic Diseases (pathology)
  • Thalamus (pathology)
  • Tomography, Emission-Computed
  • Tomography, X-Ray Computed

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