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Atrial fibrillation and acute myocardial infarction without significant coronary stenoses associated with subclinical hyperthyroidism and erythrocytosis.

Abstract
Subclinical hyperthyroidism is an increasingly recognized entity that is defined as a normal serum free thyroxine and free triiodothyronine levels with a thyroid-stimulating hormone level suppressed below the normal range and usually undetectable. It has been reported that sub-clinical hyperthyroidism is not associated with CHD or mortality from cardiovascular causes but is sufficient to induce arrhythmias including atrial fibrillation and atrial flutter. Moreover increased factor X activity in patients with subclinical hyperthyroidism represents a potential hypercoagulable state. It has been also reported an acute myocardial infarction with normal coronary arteries associated with iatrogenic hyperthyroidism and with a myocardial bridge too. It has been also reported an acute myocardial infarction without significant coronary stenoses associated with subclinical hyperthyroidism. Furthermore it has been reported that at highly increased hematocrit levels patients may experience hyperviscosity symptoms. We present a case of atrial fibrillation and acute myocardial infarction without significant coronary stenoses associated with subclinical hyperthyroidism and erythrocytosis. Also this case focuses attention on the importance of a correct evaluation of subclinical hyperthyroidism.
AuthorsSalvatore Patanè, Filippo Marte
JournalInternational journal of cardiology (Int J Cardiol) Vol. 145 Issue 1 Pg. e36-9 (Nov 05 2010) ISSN: 1874-1754 [Electronic] Netherlands
PMID19185939 (Publication Type: Case Reports, Letter)
CopyrightCopyright © 2008 Elsevier Ireland Ltd. All rights reserved.
Topics
  • Atrial Fibrillation (complications, diagnosis, physiopathology)
  • Coronary Stenosis (diagnosis, physiopathology)
  • Electrocardiography
  • Humans
  • Hyperthyroidism (complications, diagnosis, physiopathology)
  • Male
  • Middle Aged
  • Myocardial Infarction (complications, diagnosis, physiopathology)
  • Polycythemia (complications, diagnosis, physiopathology)

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