A case of living-related kidney transplantation in Bartter's syndrome.

Abstract	Bartter's syndrome is a renal tubular disorder characterized by hypokalemia, metabolic alkalosis, increased urinary excretion of potassium and prostaglandins, a relative vascular resistance to the pressor effects of exogenous angiotensin II, and hyperplasia of the juxtaglomerular apparatus. In most patients, the glomerular filtration rate is normal and chronic renal failure does not develop. We report here on a case of living-related kidney transplantation in Bartter's syndrome, in which a non-steroidal anti-inflammatory drug is suspected to be the cause of the end-stage renal disease.
Authors	J Y Kim, G A Kim, J H Song, S W Lee, J Y Han, J S Lee, M J Kim
Journal	Yonsei medical journal (Yonsei Med J) Vol. 41 Issue 5 Pg. 662-5 (Oct 2000) ISSN: 0513-5796 [Print] Korea (South)
PMID	11079628 (Publication Type: Case Reports, Journal Article)
Chemical References	Anti-Inflammatory Agents, Non-Steroidal Indomethacin
Topics	Adult Anti-Inflammatory Agents, Non-Steroidal (adverse effects) Bartter Syndrome (drug therapy) Humans Indomethacin (adverse effects) Kidney Failure, Chronic (chemically induced, surgery) Kidney Transplantation Living Donors Male

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